Understanding Spasticity After Stroke: Causes, Treatment, and What Actually Helps

NeuroRehab Team
Thursday, June 26th, 2025



Spasticity affects up to one-third of stroke survivors, turning simple daily activities into frustrating struggles against stiff, uncontrollable muscles.

Although it is one of the most common consequences of stroke, spasticity is also one of the most misunderstood and frequently mismanaged aspects of recovery.

This article explains what spasticity really is, why it happens after stroke, what the evidence says about treatment, and how patients and therapists can work together to manage it effectively.

What Is Spasticity?

Spasticity is an abnormal increase in muscle tone or stiffness that interferes with voluntary movement and can cause discomfort or pain.

After a stroke damages communication pathways in the brain, muscles may become locked in an overactive state. The normal balance between contraction and relaxation is disrupted.

For stroke survivors, spasticity commonly presents as:

  • A tight, clenched hand
  • A stiff elbow that will not fully straighten
  • A pointed foot (equinus) that drags during walking
  • Sudden involuntary muscle spasms or jerks

Spasticity is not just a movement problem. It affects hygiene, dressing, comfort, sleep, and overall independence. Without proper management, it can lead to contractures and long-term disability.

Why Does Spasticity Happen After Stroke?

Under normal conditions, the brain continuously sends signals that regulate when muscles contract and when they relax.

After a stroke, these inhibitory signals are weakened or lost. At the same time, primitive reflex pathways and spinal circuits can become overactive.

This imbalance causes normal stretch reflexes to become exaggerated. Even light touch, quick movement, or changes in position can trigger excessive muscle tightening.

Spasticity is therefore a neurological control problem, not simply a muscle problem.

How Common Is Spasticity After Stroke?

Research consistently shows that:

  • Approximately 30 to 40 percent of stroke survivors develop spasticity
  • It often emerges within the first one to three months after stroke
  • Without early intervention, spasticity frequently worsens over time

Early recognition and treatment are critical to preventing secondary complications.

How Is Spasticity Treated?

Effective spasticity management requires a multi-pronged approach. There is no single treatment that works for everyone.

The most successful strategies combine medical management, therapy, and consistent follow-through.

Stretching and Range-of-Motion Exercises

Regular, gentle stretching helps maintain joint mobility and soft tissue length.

Daily range-of-motion exercises are commonly recommended to reduce stiffness and lower the risk of contractures. While stretching alone rarely improves active function, it plays an important supportive role.

Proper Positioning and Splinting

Orthotics and splints help maintain joint alignment and prevent sustained shortening of muscles.

Night splints for the wrist, hand, or ankle are often prescribed. Proper positioning throughout the day is equally important.

Botulinum Toxin (Botox) Injections

Botulinum toxin injections are one of the most effective treatments for focal spasticity.

Botox temporarily blocks nerve signals to overactive muscles, reducing tone for several months. Strong evidence supports its use for spasticity affecting the wrist, fingers, and ankle.

Botox works best when combined with therapy that retrains movement while muscle tone is reduced.

Oral Medications

Medications such as baclofen, tizanidine, or diazepam reduce muscle tone throughout the body.

While they can be helpful for generalized spasticity, side effects such as drowsiness, weakness, and dizziness often limit long-term use.

Functional Electrical Stimulation

Functional electrical stimulation is most commonly used for foot drop or hand opening, but it may also help reduce spasticity in select cases.

By activating weak muscles and promoting more normal movement patterns, stimulation can provide inhibitory input to overactive muscles.

Physical and Occupational Therapy

Hands-on therapy remains the cornerstone of spasticity management.

Therapists focus on active movement training, task-specific practice, and teaching safe, effective use of the affected limbs.

Reducing spasticity without retraining movement rarely leads to meaningful functional gains.

Surgical Options in Severe Cases

When conservative treatments fail, surgical interventions such as tendon lengthening or intrathecal baclofen pumps may be considered.

These options are reserved for severe, refractory spasticity and require careful multidisciplinary evaluation.

What Does the Evidence Say?

Research paints a clear picture:

  • Botox has strong evidence for reducing focal spasticity and improving ease of hygiene and dressing
  • Stretching and splinting help prevent contractures but have limited impact on active function alone
  • Oral medications provide modest tone reduction but must be balanced against side effects
  • Therapy is essential for translating reduced tone into functional improvement

Lowering muscle tone is only the first step. Without follow-up training, gains do not carry over into daily life.

Key Takeaways for Patients and Caregivers

  • Do not ignore early signs of tightness or stiffness
  • Ask about Botox if spasticity interferes with daily activities or self-care
  • Stay consistent with home stretching and exercises
  • Use a combination of treatments rather than relying on a single approach

Final Thoughts: Spasticity Requires a Team Effort

Managing spasticity is about more than relaxing muscles.

It is about protecting independence, comfort, and quality of life.

With the right combination of medical treatment, skilled therapy, and active participation, stroke survivors can reduce stiffness, prevent complications, and regain meaningful control over movement.

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